{"id":1129,"date":"2023-05-07T15:03:07","date_gmt":"2023-05-07T15:03:07","guid":{"rendered":"https:\/\/medinaz.com\/blog\/?p=1129"},"modified":"2023-05-07T15:03:09","modified_gmt":"2023-05-07T15:03:09","slug":"ameloblastoma-high-yield-dental-notes","status":"publish","type":"post","link":"https:\/\/medinaz.com\/blog\/2023\/05\/07\/ameloblastoma-high-yield-dental-notes\/","title":{"rendered":"Ameloblastoma: High-yield Dental Notes"},"content":{"rendered":"\n<h2>Classification of Ameloblastoma:<\/h2>\n\n\n\n<p>Based on clinical, radiographic &amp; histopathology &amp; behavioural &amp; prognostic aspects, Ameloblastoma is <a href=\"https:\/\/www.ncbi.nlm.nih.gov\/pmc\/articles\/PMC4439660\/\" target=\"_blank\" rel=\"noreferrer noopener\">classified<\/a> as:<\/p>\n\n\n\n<ol><li>Classic- Solid\/ Multicystic ameloblastoma (SMA)<\/li><li>Unicystic ameloblastoma (UA)<\/li><li>Peripheral ameloblastoma (PA)<\/li><li>Desmoplastic ameloblastoma (DA), including hybrid lesion.<\/li><\/ol>\n\n\n\n<h2>&nbsp;Solid\/ Multicystic Ameloblastoma (SMA)<\/h2>\n\n\n\n<h3>Clinical Features:<\/h3>\n\n\n\n<ul><li>Benign epithelial tumour, with almost no tendency to metastasize. Though it is locally invasive.<\/li><li>High recurrence if improperly removed<\/li><li>Located centrally (intraosseous).<\/li><li>Few or no clinical signs in early stages.<\/li><li>Later signs:<\/li><\/ul>\n\n\n\n<ul><li>Facial deformity, loose teeth<\/li><li>Spontaneous fracture may be seen.<\/li><li>Bony swelling may be seen<\/li><li>Pain: Due to either pressure of growing tumour on nerves or secondary infection.<\/li><\/ul>\n\n\n\n<ul><li>Enlarging tumour makes the surrounding bone elicit<strong> Crepitation\/ Egg shell crackling<\/strong><\/li><li>Perforation of bone may occur.<\/li><\/ul>\n\n\n\n<h3>Radiographic features:<\/h3>\n\n\n\n<ul><li>Multilocular lesion- typical appearance<\/li><li>Unilocular may also be seen.<\/li><li>Unilocular:<\/li><\/ul>\n\n\n\n<ul><li>Well defined single radiolucency.<\/li><li>If associated with unerupted tooth, may resemble<strong> Dentigerous cyst or OKC.<\/strong><\/li><\/ul>\n\n\n\n<h3>Epidemiology:<\/h3>\n\n\n\n<ul><li><strong>Black<\/strong>&gt; White<\/li><li><strong>Age<\/strong>&#8211; 35-37 yrs average age at time of diagnosis.<\/li><li>According to<strong> Gardener<\/strong>, mean age at time of diagnosis:<\/li><\/ul>\n\n\n\n<ul><li><strong>SMA<\/strong>&#8211; about 39 yrs<\/li><li><strong>Unicystic<\/strong>&#8211; about 22 yrs<\/li><li><strong>Peripheral<\/strong>&#8211; about 51 yrs<\/li><\/ul>\n\n\n\n<ul><li><strong>Gender<\/strong>&#8211; almost equal or slight male (1.1:1) predilection.<\/li><li><strong>Location<\/strong>: Mandible&gt; Maxilla (2.2:1)<\/li><li>Posterior mandible&gt; anterior mandible&gt; posterior maxilla&gt; anterior maxilla<\/li><\/ul>\n\n\n\n<h3>Pathology:<\/h3>\n\n\n\n<h4><strong>Pathogenesis:<\/strong><\/h4>\n\n\n\n<ul><li>Mostly arise from <strong>odontogenic epithelial remnants<\/strong>, specifically remnants of <strong>dental lamina<\/strong>.<\/li><li>If these remnants lie outside bone in soft tissues, they form<strong> Peripheral Ameloblastoma.<\/strong><\/li><li>May arise as neoplastic change in lining or wall of cysts like Dentigerous or OKC called<strong> Mural ameloblastoma.<\/strong> (usually seen in posterior region).<\/li><li>May originate from<strong> Epithelial rests of Malassez<\/strong>.<\/li><\/ul>\n\n\n\n<h4>Microscopy:<\/h4>\n\n\n\n<p><strong>WHO 1992 definition: <\/strong>A polymorphic neoplasm consisting of proliferating odontogenic epithelium which usually has a follicular or plexiform pattern lying in a fibrous stroma.<\/p>\n\n\n\n<p>The various histologic patterns of SMA include:<\/p>\n\n\n\n<p><strong>Follicular pattern<\/strong><\/p>\n\n\n\n<ul><li>Epithelial Islands: Contain central mass of polyhedral cells, or loosely connected angular cells resembling stellate reticulum.<\/li><li>Peripheral cells in the epithelial islands are cuboidal or columnar resembling inner enamel epithelium or pre-ameloblast.<\/li><li>Cystic degeneration is common within epithelial islands<\/li><\/ul>\n\n\n\n<p><strong>Plexiform pattern<\/strong><\/p>\n\n\n\n<ul><li>Tumour epithelium arranged as a network<\/li><li>This network bound by cuboidal to columnar cells &amp; includes <strong>Stellate Reticulum <\/strong>like cells.<\/li><li><strong>Cyst formation <\/strong>is due to stromal degeneration may be seen.<\/li><li><strong>Hyaline bodies like odontogenic cyst epithelium\/wall may be seen<\/strong><\/li><\/ul>\n\n\n\n<p><strong>Acanthomatous SMA:<\/strong><\/p>\n\n\n\n<ul><li>Extensive squamous metaplasia<\/li><li>Keratin may be formed sometimes (within tumour island)<\/li><li>Generally, shows<strong> follicular<\/strong> pattern<\/li><li><strong>Third most common <\/strong>histologic type.<\/li><\/ul>\n\n\n\n<p><strong>Granular cell SMA:<\/strong><\/p>\n\n\n\n<ul><li>Most often it shows <strong>Follicular<\/strong> pattern.<\/li><li><strong>Granular transformation<\/strong> of central <strong>stellate cells.<\/strong><\/li><li>Granular cells may be <strong>Cuboidal<\/strong>, <strong>Columnar<\/strong> or <strong>Round<\/strong>.<\/li><li>Cytoplasm filled with<strong> Acidophilic granules.<\/strong><\/li><li>Granularity may be due to <strong>increased apoptosis<\/strong> &amp; <strong>phagocytosis<\/strong> of cells by neighbouring <strong>neoplastic cells<\/strong>.<\/li><\/ul>\n\n\n\n<p><strong>Desmoplastic SMA:<\/strong><\/p>\n\n\n\n<ul><li>Usually <strong>follicular<\/strong> pattern of SMA<\/li><li>Connective tissue shows marked <strong>hyalinization<\/strong> (desmoplasia).<\/li><\/ul>\n\n\n\n<p><strong>Basal cell SMA:<\/strong><\/p>\n\n\n\n<ul><li>Resemble <strong>basal<\/strong> &amp; <strong>suprabasal<\/strong> <strong>spinosum cells.<\/strong><\/li><li><strong>Rare<\/strong> variant<\/li><li>SMA shows predominant <strong>basaloid pattern.<\/strong><\/li><li>Most <strong>actively proliferating<\/strong> type. It shows <strong>positive labelling<\/strong> for both<strong><em>PCNA &amp; KP-67.<\/em><\/strong>&nbsp;<\/li><li>This variant has <strong>most immature cells<\/strong>.<\/li><\/ul>\n\n\n\n<p><strong>Clear cell SMA:<\/strong><\/p>\n\n\n\n<ul><li>Clear cells in <strong>stellate cell area<\/strong> of SMA follicles<\/li><li><strong>PAS<\/strong> positive<\/li><li>May show <strong>malignant transformation.<\/strong><\/li><\/ul>\n\n\n\n<p>K<strong>eratoameloblastoma(KA) &amp; papilliferous KA:<\/strong><\/p>\n\n\n\n<ul><li>Simultaneous occurrence of areas of <strong>Ameloblastoma<\/strong> with pronounced <strong>keratinization<\/strong> &amp; <strong>cystic areas<\/strong> of resembling OKC.<\/li><li>Extremely <strong>rare<\/strong>.<\/li><\/ul>\n\n\n\n<p><strong>Connective tissue of all histologic variants of SMA:<\/strong><\/p>\n\n\n\n<ul><li>Contains fibroblasts, collagen fibres &amp; myofibroblasts.<\/li><\/ul>\n\n\n\n<h3>Treatment:<\/h3>\n\n\n\n<ul><li>Treatment ranges from <strong>simple enucleation<\/strong> and <strong>curettage<\/strong> to <strong>en bloc resection<\/strong>.<\/li><li><strong>Curettage only<\/strong>, often leave small islands of tumor within the bone, this results in <strong>recurrences (50-90%).<\/strong><\/li><li><strong>Marginal resection:<\/strong> Most <strong>widely used<\/strong> treatment, but <strong>recurrence<\/strong> of up to <strong>15%<\/strong> may be seen.<\/li><li><strong>Removal of the tumor, followed by peripheral ostectomy<\/strong>, often reduces the need for extensive reconstructive surgery.<\/li><li>Ameloblastomas of the <strong>posterior maxilla<\/strong> are particularly <strong>dangerous<\/strong> because of the <strong>difficulty<\/strong> of obtaining an <strong>adequate surgical margin<\/strong> around the tumor.<\/li><li>Though <strong>ameloblastoma<\/strong> is <strong>radiosensitive<\/strong>, Radiation therapy is <strong>contraindicated<\/strong> because of a possibility of <strong>secondary radiation-induced malignancy<\/strong>.<\/li><\/ul>\n\n\n\n<h2>Unicystic Ameloblastoma<\/h2>\n\n\n\n<ul><li><strong>Well defined<\/strong>, often large <strong>monocystic<\/strong> cavity with a lining.<\/li><li>Locally but rarely entirely <strong>lined<\/strong> by <strong>odontogenic epithelium<\/strong>.<\/li><li>On the<strong> Luminal surface<\/strong> of cyst, one or several <strong>polypoid<\/strong> or <strong>papillomatous<\/strong>, <strong>pedunculated<\/strong> exophytic <strong>masses<\/strong> may be seen: These are <strong>called<\/strong> <strong>Intracystic<\/strong>, <strong>Luminal<\/strong> or <strong>Intraluminal<\/strong> ameloblastoma.<\/li><li>Epithelial <strong>nodules<\/strong> may grow <strong>within<\/strong> the <strong>connective tissue:<\/strong> Called <strong>Mural<\/strong> or <strong>Intramural.<\/strong><\/li><li>May be associated with an <strong>unerupted tooth<\/strong>.<\/li><\/ul>\n\n\n\n<h3>Clinical &amp; Radiographic findings:<\/h3>\n\n\n\n<ul><li>If there is <strong>secondary infection:<\/strong><\/li><\/ul>\n\n\n\n<ul><li>Local <strong>swelling<\/strong><\/li><li>Occasional <strong>pain<\/strong><\/li><li>Lip <strong>numbness<\/strong><\/li><li><strong>Discharge<\/strong> or drainage<\/li><\/ul>\n\n\n\n<ul><li>May be <strong>unilocular<\/strong> or <strong>multilocular; <\/strong>Unilocular more common<\/li><li>Root resorption is common.<\/li><li>If <strong>crown<\/strong> of unerupted tooth is <strong>involved<\/strong>, it is <strong>displaced<\/strong> by cystic tumour rather than project into cystic lumen \u2013 <strong>Differentiation<\/strong> from <strong>Dentigerous cyst<\/strong>.<\/li><\/ul>\n\n\n\n<h3>Epidemiology:<\/h3>\n\n\n\n<ul><li><strong>Tooth associated<\/strong> lesion occurs in <strong>younger patients<\/strong> compared to those not associated with tooth.&nbsp;<\/li><li><strong>Males:<\/strong> <strong>Tooth associated<\/strong> lesion more common; <strong>Females:<\/strong> Lesion <strong>without tooth<\/strong> are common.<\/li><li><strong>Mandible<\/strong> &gt; Maxilla; <strong>Posterior mandible<\/strong> &amp; <strong>ascending ramus<\/strong> is most commonly involved.<\/li><\/ul>\n\n\n\n<h3>Pathology:<\/h3>\n\n\n\n<h4>Pathogenesis:<\/h4>\n\n\n\n<ul><li>May arise from <strong>pre-existing odontogenic cyst<\/strong> (commonly <strong>Dentigerous cyst<\/strong>) or it may arise <strong>de novo.<\/strong><\/li><li>According to <strong>Leider et al.:<\/strong> They may arise from:<\/li><\/ul>\n\n\n\n<ol><li>From <strong>REE.<\/strong><\/li><li>From <strong>Dentigerous<\/strong> or another <strong>odontogenic cyst.<\/strong><\/li><li>From <strong>Solid ameloblastoma<\/strong> undergoing cystic degeneration.<\/li><\/ol>\n\n\n\n<h4>Microscopy:&nbsp;<\/h4>\n\n\n\n<ul><li><strong>Minimum criteria<\/strong> for diagnosing a lesion a unicystic ameloblastoma:<\/li><\/ul>\n\n\n\n<ul><li>Single <strong>cystic sac<\/strong>.<\/li><li>Lined by <strong>odontogenic epithelium<\/strong> (usually <strong>present locally<\/strong>).<\/li><li><strong>Epithelium<\/strong> may be variable, <strong>mimicking<\/strong> lining of <strong>Dentigerous<\/strong> or <strong>Radicular cyst.<\/strong><\/li><\/ul>\n\n\n\n<p>Sub-groups of unicystic ameloblastoma:<\/p>\n\n\n\n<p><strong>Luminal type:<\/strong><\/p>\n\n\n\n<ul><li><strong>Epithelial lining<\/strong> may show <strong>transformation<\/strong> to <strong>cuboidal or columnar<\/strong> basal cells.<\/li><li><strong>Nuclear palisading<\/strong> with <strong>polarization<\/strong>.<\/li><li>Cytoplasmic <strong>vacuolization<\/strong>.<\/li><li>Intercellular <strong>spacing<\/strong>.<\/li><li>Sub-epithelial <strong>hyalinization<\/strong>.<\/li><\/ul>\n\n\n\n<p><strong>Intramural tissue:<\/strong><\/p>\n\n\n\n<ul><li><strong>Infiltration<\/strong> from <strong>cyst lining<\/strong> or as <strong>free islands<\/strong> of follicles (SMA) often with <strong>central cystic degeneration.<\/strong><\/li><li><strong>About 2\/3<\/strong> of both tooth-associated &amp; non-tooth-associated show <strong>intramural invasion.<\/strong><\/li><li>Though slightly <strong>more common<\/strong> in <strong>non-tooth type.<\/strong><\/li><li><strong>Intra luminal<\/strong> proliferation <strong>more common<\/strong> in <strong>tooth associated<\/strong> type.<\/li><li>Tumours with <strong>intramural invasion<\/strong> have <strong>higher recurrence.<\/strong><\/li><\/ul>\n\n\n\n<h3>Treatment:<\/h3>\n\n\n\n<ul><li>May be treated <strong>conservatively by enucleation<\/strong> or it may need <strong>aggressive treatment as classic SMA<\/strong>, depending on the <strong>histopathologic presentation<\/strong>.<\/li><\/ul>\n\n\n\n<h2>Peripheral Ameloblastoma:<\/h2>\n\n\n\n<ul><li>Peripheral ameloblastoma is a <strong>benign neoplasm<\/strong> or <strong>hamartomatous<\/strong> lesion confined to <strong>soft tissue<\/strong> overlying tooth bearing area of jaws.<\/li><li>Several <strong>characteristic similar to SMA.<\/strong><\/li><li><strong>Do not invade<\/strong> underlying bone.<\/li><\/ul>\n\n\n\n<h3>Clinical &amp; Radiographic features:<\/h3>\n\n\n\n<ul><li><strong>Painless<\/strong>, <strong>sessile<\/strong>, <strong>firm<\/strong> &amp; <strong>exophytic<\/strong> growth.<\/li><li><strong>Surface<\/strong> is relatively <strong>smooth<\/strong>, but may be <strong>granular<\/strong> or <strong>pebbly<\/strong>. Sometimes <strong>Papillary<\/strong> or <strong>Warty<\/strong>.<\/li><li><strong>Surface colour:<\/strong>&nbsp; Normal to <strong>red<\/strong> to <strong>dark red<\/strong>.<\/li><li><strong>Size<\/strong>&#8211; 0.3 to 4.5 cm (1.3 cm)<\/li><li><strong>Superficial bone erosion<\/strong> may be seen, due to <strong>pressure<\/strong> of lesion.<\/li><\/ul>\n\n\n\n<h3>Epidemiology:<\/h3>\n\n\n\n<ul><li>2-10% of all Ameloblastomas.<\/li><li><strong>Age<\/strong>&#8211; <strong>9-92<\/strong> yrs. (<strong>Average<\/strong> age: <strong>52 yrs.). More common<\/strong> in 5<sup>th<\/sup> to 7<sup>th<\/sup> decade.<\/li><li><strong>Gender:<\/strong> more common in <strong>males<\/strong>.<\/li><li><strong>Location: <\/strong>more common in <strong>mandible. Mandibular premolar region<\/strong> most common site.<\/li><\/ul>\n\n\n\n<h3>Pathology:<\/h3>\n\n\n\n<h4>Pathogenesis:<\/h4>\n\n\n\n<ul><li>Probably arises from <strong>dental lamina<\/strong> remnants. (<strong>cell rests of Serre<\/strong>).<\/li><li>May also arise from<strong> surface epithelium.<\/strong> <strong>Continuity<\/strong> between <strong>tumour<\/strong> &amp; <strong>surface epithelium<\/strong> has been seen.<\/li><\/ul>\n\n\n\n<h4>Microscopy:<\/h4>\n\n\n\n<p><strong>Epithelium:&nbsp;<\/strong><\/p>\n\n\n\n<ul><li>Epithelial islands show <strong>palisaded columnar basal<\/strong> cells.<\/li><li><strong>Stellate reticulum like cells<\/strong> are few in number (negligible).<\/li><li>Lesions exhibiting <strong>Acanthomatous areas<\/strong> are difficult to distinguise from <strong>Basal cell carcinoma.<\/strong><\/li><li><strong>Ghost cells<\/strong> in Acanthomatous area may be seen. May be confused with<strong> Calcifying ghost cell odontogenic cyst.<\/strong><\/li><li><strong>Vacuolated<\/strong> or <strong>clear cells<\/strong> may be seen in some parts of tumour as clusters.<\/li><\/ul>\n\n\n\n<p><strong>Stroma:<\/strong>&nbsp;<\/p>\n\n\n\n<ul><li><strong>Mature<\/strong> fibrous connective tissue (<strong>calcification<\/strong> may be seen <strong>sometimes<\/strong>).<\/li><\/ul>\n\n\n\n<h3>Treatment:<\/h3>\n\n\n\n<ul><li>It exhibits a <strong>milder biologic behaviour<\/strong> than the SMA. So, <strong>wide excision<\/strong> is usually <strong>not needed.<\/strong><\/li><li>Conservative <strong>supra-periosteal surgical excision<\/strong> with adequate <strong>disease-free margins<\/strong>.<\/li><li><strong>Recurrence<\/strong> rate <strong>lower than SMA.<\/strong><\/li><\/ul>\n\n\n\n<h2>Desmoplastic Ameloblastoma<\/h2>\n\n\n\n<ul><li>Extensive <strong>stromal collagenisation<\/strong> or <strong>desmoplasia<\/strong>.<\/li><li><strong>Hybrid ameloblastoma:<\/strong> Shows features of both <strong>Desmoplastic variant<\/strong> &amp; <strong>classic follicular<\/strong> ameloblastoma. It is probably a <strong>transitional form<\/strong> of desmoplastic ameloblastoma.<\/li><\/ul>\n\n\n\n<h3>Clinical features:<\/h3>\n\n\n\n<ul><li><strong>Benign<\/strong>, locally <strong>infiltrative<\/strong>, <strong>epithelial<\/strong> neoplasm.<\/li><li>A variant or <strong>sub-type of SMA.<\/strong><\/li><li><strong>Painless<\/strong> <strong>swelling:<\/strong> chief complaint in most cases.<\/li><\/ul>\n\n\n\n<h3>Radiographic features:<\/h3>\n\n\n\n<ul><li><strong>Well defined borders<\/strong> are usually <strong>not seen.<\/strong><\/li><li><strong>Mixed radiolucent\/ radiopaque<\/strong> in most cases.<\/li><li><strong>Root resorption<\/strong> is common.<\/li><li><strong>New bone formation<\/strong> may be seen.<\/li><li>Shows <strong>infiltration in adjacent bone marrow<\/strong> space. It causes <strong>ill-defined borders<\/strong> of lesion.<\/li><\/ul>\n\n\n\n<h3>Epidemiology:<\/h3>\n\n\n\n<ul><li><strong>4-13%<\/strong> of all SMA<\/li><li>Age \u2013 <strong>17-72yrs<\/strong> (43yrs)<\/li><li>Slight or no <strong>male predominance.<\/strong><\/li><li>Occurs with <strong>almost equal frequency<\/strong> in <strong>both jaws<\/strong>. More common in <strong>Anterior region.<\/strong><\/li><\/ul>\n\n\n\n<h3>Pathogenesis:<\/h3>\n\n\n\n<h4>Microscopy:<\/h4>\n\n\n\n<ul><li>Consists of <strong>proliferating, irregular<\/strong>, often <strong>bizarrely<\/strong> shaped <strong>islands and cords<\/strong> of <strong>odontogenic epithelium<\/strong> of varying sizes embedded in a <strong>desmoplastic connective tissue<\/strong> stroma.<\/li><\/ul>\n\n\n\n<p><strong>Epithelial islands:&nbsp;<\/strong><\/p>\n\n\n\n<ul><li><strong>Irregular<\/strong> in shape and have a <strong>pointed stellate<\/strong> appearance.<\/li><li>Pathognomonic <strong>animal like outline.<\/strong><\/li><li><strong>Peripheral<\/strong> epithelial <strong>cells<\/strong> are usually <strong>cuboidal<\/strong>, rarely columnar with <strong>reversal of polarity.<\/strong><\/li><li><strong>Hyperchromatic nuclei<\/strong> may be seen sometimes.<\/li><\/ul>\n\n\n\n<p><strong>Centre of epithelial islands:<\/strong><\/p>\n\n\n\n<ul><li><strong>Hypercellular<\/strong> with <strong>spindle shaped or squamatoid<\/strong> or rarely <strong>keratinized epithelial cells<\/strong>.<\/li><li><strong>Microcysts<\/strong> containing <strong>eosinophilic amorphous deposits<\/strong> or empty are common within tumour islands.<\/li><li>Foci of <strong>keratinization<\/strong> may be seen sometimes.<\/li><li><strong>Glandular differentiation<\/strong> with <strong>mucous cells<\/strong> formation may be seen in tumour nests.<\/li><\/ul>\n\n\n\n<p><strong>Connective tissue stroma:<\/strong><\/p>\n\n\n\n<ul><li>Extensive <strong>stromal dysplasia.<\/strong><\/li><li><strong>Moderately cellular<\/strong>, fibrous connective tissue.<\/li><li><strong>Collagen fibres<\/strong> are <strong>thick &amp; numerous<\/strong>. They appear to compress tumour islands.<\/li><li><strong>Myxoid changes<\/strong> may be seen in stroma <strong>around odontogenic epithelium<\/strong>.<\/li><li><strong>Metaplastic bone<\/strong> formation may be seen.<\/li><li><strong>Capsule<\/strong> \u2013 peripheral fibrous condensation <strong>not always seen.<\/strong><\/li><\/ul>\n\n\n\n<h3>Treatment:<\/h3>\n\n\n\n<p>Same as SMA &nbsp; &nbsp; &nbsp; &nbsp;<\/p>\n\n\n\n<p> &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp;<\/p>\n\n\n\n<figure class=\"wp-block-image size-large is-style-default\"><img loading=\"lazy\" width=\"1024\" height=\"791\" src=\"https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Notes-1-1024x791.jpg\" alt=\"Ameloblastoma Notes 1\" class=\"wp-image-1132\" srcset=\"https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Notes-1-1024x791.jpg 1024w, https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Notes-1-300x232.jpg 300w, https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Notes-1-768x593.jpg 768w, https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Notes-1-260x200.jpg 260w, https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Notes-1-87x67.jpg 87w, https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Notes-1.jpg 1116w\" sizes=\"(max-width: 1024px) 100vw, 1024px\" \/><figcaption><strong><span class=\"has-inline-color has-black-color\">Ameloblastoma Notes 1<\/span><\/strong><\/figcaption><\/figure>\n\n\n\n<figure class=\"wp-block-image size-large is-style-default\"><img loading=\"lazy\" width=\"1024\" height=\"791\" src=\"https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Notes-2-1024x791.jpg\" alt=\"Ameloblastoma Notes 2\" class=\"wp-image-1133\" srcset=\"https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Notes-2-1024x791.jpg 1024w, https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Notes-2-300x232.jpg 300w, https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Notes-2-768x593.jpg 768w, https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Notes-2-260x200.jpg 260w, https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Notes-2-87x67.jpg 87w, https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Notes-2.jpg 1116w\" sizes=\"(max-width: 1024px) 100vw, 1024px\" \/><figcaption><strong><span class=\"has-inline-color has-black-color\">Ameloblastoma Notes 2<\/span><\/strong><\/figcaption><\/figure>\n\n\n\n<figure class=\"wp-block-image size-large is-style-default\"><img loading=\"lazy\" width=\"1024\" height=\"791\" src=\"https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Notes-3-1024x791.jpg\" alt=\"Ameloblastoma Notes 3\" class=\"wp-image-1134\" srcset=\"https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Notes-3-1024x791.jpg 1024w, https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Notes-3-300x232.jpg 300w, https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Notes-3-768x593.jpg 768w, https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Notes-3-260x200.jpg 260w, https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Notes-3-87x67.jpg 87w, https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Notes-3.jpg 1116w\" sizes=\"(max-width: 1024px) 100vw, 1024px\" \/><figcaption><strong><span class=\"has-inline-color has-black-color\">Ameloblastoma Notes 3<\/span><\/strong><\/figcaption><\/figure>\n\n\n\n<figure class=\"wp-block-image size-large is-style-default\"><img loading=\"lazy\" width=\"1024\" height=\"791\" src=\"https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Notes-4-1024x791.jpg\" alt=\"Ameloblastoma Notes 4\" class=\"wp-image-1135\" srcset=\"https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Notes-4-1024x791.jpg 1024w, https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Notes-4-300x232.jpg 300w, https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Notes-4-768x593.jpg 768w, https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Notes-4-260x200.jpg 260w, https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Notes-4-87x67.jpg 87w, https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Notes-4.jpg 1116w\" sizes=\"(max-width: 1024px) 100vw, 1024px\" \/><figcaption><strong><span class=\"has-inline-color has-black-color\">Ameloblastoma Notes 4<\/span><\/strong><\/figcaption><\/figure>\n\n\n\n<figure class=\"wp-block-image size-large is-style-default\"><img loading=\"lazy\" width=\"1024\" height=\"1024\" src=\"https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Dental-tumor-book--1024x1024.jpg\" alt=\"Dental tumor book\" class=\"wp-image-1138\" srcset=\"https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Dental-tumor-book--1024x1024.jpg 1024w, https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Dental-tumor-book--300x300.jpg 300w, https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Dental-tumor-book--150x150.jpg 150w, https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Dental-tumor-book--768x768.jpg 768w, https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Dental-tumor-book--1536x1536.jpg 1536w, https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Dental-tumor-book--370x370.jpg 370w, https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Dental-tumor-book--45x45.jpg 45w\" sizes=\"(max-width: 1024px) 100vw, 1024px\" \/><figcaption><strong><span class=\"has-inline-color has-black-color\">Dental tumor book<\/span><\/strong><\/figcaption><\/figure>\n\n\n\n<p>&#8220;High Yield Visual Book of Dental Cyst&#8221; is now available on &#8220;Medinaz&#8221; App. The App is available on Appstore &amp; Playstore. Visit our website <a href=\"https:\/\/medinaz.com\" target=\"_blank\" rel=\"noreferrer noopener\">www.medinaz.com<\/a> for other available books.<\/p>\n\n\n\n<p><strong>Book overview:<\/strong><br><strong>&#8211; All the necessary High-Yield Points<\/strong><br><strong>&#8211; 550+ Frequently tested facts<\/strong><br><strong>&#8211; 300+ hand drawn Images<\/strong><br><strong>&#8211; Mnemonics to remember<\/strong><br><strong>&#8211; Helpful for: &nbsp;NBDE, NEET MDS, and Board exams<\/strong><br><strong>&#8211; FREE UPDATES up to 1 year from the date of publish<\/strong><br><strong>&#8211;&nbsp;(Time span to be counted from the day it was published)<\/strong><br><strong>&#8211; Neatly&nbsp;organized materials<\/strong><br><strong>&#8211; Lifetime access<\/strong><br><strong>&#8211; Format Image based PDF<\/strong><\/p>\n\n\n\n<p><\/p>\n\n\n\n<p>Check other Dental Notes: <strong><a href=\"https:\/\/medinaz.com\/blog\/category\/dental-notes\/\" target=\"_blank\" rel=\"noreferrer noopener\">Click here<\/a><\/strong><\/p>\n","protected":false},"excerpt":{"rendered":"<p>Classification of Ameloblastoma: Based on clinical, radiographic &amp; histopathology &amp; behavioural &amp; prognostic aspects, Ameloblastoma is classified as: Classic- Solid\/ Multicystic ameloblastoma (SMA) Unicystic ameloblastoma (UA) Peripheral ameloblastoma (PA) Desmoplastic ameloblastoma (DA), including hybrid lesion. &nbsp;Solid\/ Multicystic Ameloblastoma (SMA) Clinical Features: Benign epithelial tumour, with almost no tendency to metastasize. Though it is locally invasive.<\/p>\n","protected":false},"author":1,"featured_media":1142,"comment_status":"closed","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"ub_ctt_via":""},"categories":[45,53],"tags":[216,75],"featured_image_src":"https:\/\/medinaz.com\/blog\/wp-content\/uploads\/2023\/05\/Ameloblastoma-Highyield-Notes.jpg","author_info":{"display_name":"Medinaz Academy","author_link":"https:\/\/medinaz.com\/blog\/author\/medinaz-blog-admin\/"},"yoast_head":"<!-- This site is optimized with the Yoast SEO plugin v19.4 - https:\/\/yoast.com\/wordpress\/plugins\/seo\/ -->\n<title>Ameloblastoma: High-yield Dental Notes - Medinaz Blog<\/title>\n<meta name=\"description\" content=\"This dental notes contains all the necessary high-yield points about Ameloblastoma. 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